Nature Portfolio

Nature Portfolio

Book and Periodical Publishing

London, Greater London 243,607 followers

About us

Springer Nature publishes journals, databases and online products and services across the life, physical and applied sciences and, most recently, clinical medicine. Content encompasses daily news from award-winning journalists, expert opinion and practical methodology, and more high impact research and reviews than any science publisher. Over 70 journals are published, some in association with prestigious academic societies. Nature Portfolio provides news content through Nature News. Scientific career information and free job postings are offered on Naturejobs. Nature was first published in 1869. Springer Nature has its principal offices in London, New York and Tokyo with offices in offices in Basingstoke, Boston, Buenos Aires, Delhi, Hong Kong, Madrid, Melbourne, Munich, Paris, San Francisco, Seoul and Washington DC.

Website
https://www.nature.com/nature-portfolio
Industry
Book and Periodical Publishing
Company size
501-1,000 employees
Headquarters
London, Greater London
Type
Privately Held
Specialties
Publishing and Science Publishing

Locations

  • Primary

    The Macmillan Building

    4 Crinan Street

    London, Greater London N1 9XW, GB

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Employees at Nature Portfolio

Updates

  • Nature Portfolio reposted this

    View profile for David Payne, graphic

    Managing Editor, Careers and Supplements, Nature at Springer Nature Group

    "Do fewer things, work at a natural pace, obsess over quality." But can #academics today apply "slow productivity" as espoused by computer scientist Cal Newport in his latest book? Thanks Cal, Maya Gosztyla and Megan Rogers, for sharing perspectives with Anne Gulland and Nature Magazine.

    Slow productivity worked for Marie Curie — here’s why you should adopt it, too

    Slow productivity worked for Marie Curie — here’s why you should adopt it, too

    nature.com

  • Nature Portfolio reposted this

    Nature Neuroscience publishes a special issue on glia! Our August issue just went live, and it's a special issue on glia. Glia is a blanket term that refers to several very different cell-types: astrocytes, microglia and oligodendrocytes (as well as their antecedents, oligodendrocyte precursor cells). Research into glia has become one of the most exciting and dynamic subfields of neuroscience, yet there is still much to be discovered about the diverse forms and functions of these cells. As the editors of Nature Neuroscience, we have been exhilarated by the pace of discoveries in this area and are delighted to showcase some of this transformative work in our pages. This month’s issue represents a celebration of glia from start to finish. We begin by highlighting some exciting papers about glia that have been published in other journals in recent weeks. We share conversations with established leaders and emerging voices in the field, Lucas Cheadle, Sonia Mayoral, Beth Stevens and Andrea Volterra. A Review from Lindsay Osso and Ethan Hughes challenges the dogmatic view of myelin as static and illuminates what is known about its renewal and remodeling. We also present primary research papers that reflect the breadth of the glial research field. Tewari et al. describe how astrocytic regulation of synapses depends on perineuronal nets, and O’Shea et al. characterize border-forming wound-repair astrocytes that emerge following spinal cord injury or stroke. Foerster et al. demonstrate a role for oligodendrocyte heterogeneity — specifically cells of dorsal versus ventral origin — in brain development, and we learn about roles for oligodendrocyte lineage cells in diseases that have mostly been thought of as involving neurons: neurofibromatosis type 1 and prion-induced neurodegeneration. Three papers examine interactions between microglia and other glial cell types: Kedia, Ji and colleagues reveal how CD8+ T cells trigger microglia to damage myelin in a mouse model of Alzheimer’s disease; Chen, Luan, Liu and colleagues describe how brain injury causes astrocytic ATP release (an ‘inflare’) that in turn signals to microglia; and Huang, Wang and colleagues examine how plexin-B1 expressed in periplaque reactive astrocytes in a mouse model of Alzheimer’s disease restricts access of microglia to amyloid plaques. Brown et al. even describe some circuit maturation processes that do not require microglia. And, rest assured, we have some compelling papers for our neuron-loving readers, too.

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